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Therapeutic strategies are in early stages of development. The most common systemic amyloidosis in the developed world is AL or primary amyloidosis, caused by clonal immunoglobulin light-chain deposition in the developing world, serum amyloid A protein type [AA] amyloidosis, secondary to chronic infections and inflammation, occurs more frequently ; . AL amyloidosis has been considered an orphan disease, although its incidence is about one-third that of multiple myeloma, and some studies of myeloma patients have suggested that concomitant AL amyloidosis is quite!
Instead of springing back when pulled, the skin sags. Additionally, the muscle and tissue composition under the skin changes, leading to deeper lines and wrinkles. Although it is not possible to turn back the hands of time, effects can be dramatically slowed down and improved with the arsenal of anti-aging treatments and medical-grade skin care products. Using UV sun protection with an SPF of at least 30 is the most important weapon needed to combat the aging process. A skincare protocol is necessary to help reverse aging and maintain anti- aging remedies. This includes a treatment plan combined with a home skin care program. There is no one right way to correct aging facial skin as we all have a different set of genetic triggers and what may work well for one person may not work well for another. Our job as physicians and medical aestheticians is to determine a treatment plan to get the best possible results for each patient. Mention this Newsletter for a complimentary mini facial and skin care analysis. Call 770-457-6303 more about skin care.
American Association for State and Local History ABBR: AASLH BT: organizations n., AASLH, abbr. ~ An association that supports organizations which preserve and interpret American history in states and communities, as well as the staff and volunteers who work in those organizations. Notes Although principally an association of historical societies, AASLH is concerned with archives because these organizations typically hold archival collections. See : aaslh . Citations The American Association for State and Local History provides leadership, service, and support for its members, who preserve and interpret state and local history in order to make the past more meaningful in American Society. [American Association for State and Local History 4 ; ] American Institute for the Conservation of Historic and Artistic Works ABBR: AIC n., AIC, abbr. ~ A national organization for professionals working in the field of preserving historical and artistic works. Notes Members include practicing conservators, conservation scientists, educators, administrators, collections care professionals, technicians, and students; archivists, curators, and other museum and library professionals; architects and art historians; and individuals from related disciplines. See : aic anford . American Library Association ABBR: ALA RT: Rare Book and Manuscript Section n., ALA, abbr. ~ An organization that promotes the value of libraries and provides support for the professionals, staff, and volunteers who work in the field of librarianship. Notes The mission of the ALA is to provide leadership for the development, promotion and improvement of library and information services and the profession of librarianship in order to enhance learning and ensure access to information for all. ALA is committed to focus its energy and resources in five key action areas: diversity, education and continuous learning, equity of access, intellectual freedom, and 21st century literacy. Members work in academic, public, school, government, and special libraries. See : ala . American National Standards Institute ABBR: ANSI BT: organizations n., ANSI, abbr. ~ A non-governmental organization chartered to coordinate the development of standards within the United States and to harmonize national standards with international standards with the International Organization for Standardization ISO ; . Notes See : ansi . ANSI standards are entered under their full name, with a cross reference from the code. For example, the International Standard for Serial Numbers ISSN ; is found under that name, with a see reference from Z39.9.
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18.2 BTG Israel shall have no liability for any loss to Raw Materials or Product stored by BTG Israel pursuant to Article II, unless caused by BTG Israel's negligence. 18.3 Each Party hereto agrees to promptly notify the other Party of any event of force majeure under Section 18.1 above and to employ all reasonable efforts toward prompt resumption of its performance hereunder when possible if such performance is delayed or interrupted by reason of such event. ARTICLE XIX - NOTICES 19.1 All notices and other communications required or desired to be given or sent by one party to the other party shall be in writing, in the English language, and shall be deemed to have been given a ; on the date of delivery, if delivered to the persons identified below, b ; five calendar days after mailing if mailed, with proper postage, by certified or registered airmail, postage prepaid, return receipt requested, addressed as set forth below, c ; on the date of receipt if sent by telex or telecopy, and confirmed in writing in the manner set forth in b ; on before the next day after the sending of the telex or telecopy, or d ; two business days after delivered to an internationally recognized overnight courier service marked for overnight delivery, as follows.
Cardiac hypertrophy, an increase in the size and or thickness of the ventricles in the heart, is an important compensatory mechanism for pathophysiological states and an independent predictor of cardiovascular morbidity and mortality 1, 2 ; . Several studies have shown that left ventricular mass in healthy populations and athletes is at least partially genetically determined 35 ; . At the cellular level, cardiac hypertrophy results largely from an increase in the size of individual cardiac myocytes. A number of molecular pathways related to hypertrophy have been studied in vitro and in animal models, including the alpha- and beta-adrenergic nervous systems, the renin-angiotensin-aldosterone system, endothelin-1, and calcineurin 6, 7 ; . Pharmacological blockade of these pathways, to varying degrees, has proven beneficial in the treatment of cardiovascular disorders, including heart failure. Polymorphisms in the adrenergic nervous system and the renin-angiotensin-aldosterone system have been reported to affect ventricular mass and or hypertrophy, although the findings remain controversial 3, 8, 9 ; . See page 499 Atrial natriuretic peptide ANP ; and brain natriuretic peptide BNP ; are produced by and stored in atrial and ventricular myocytes, respectively 10, 11 ; . Both peptides are released in response to stretch and cleaved into an N-terminal peptide fragment N-terminal proANP, N-terminal pro-BNP ; and the active peptide. The peptides bind to their receptors mainly natriuretic peptide receptor A [NPRA], but also NPRB and NPRC ; , lead to vasodilation and natriuresis, and are degraded by neutral endopeptidase NEP ; . Measurements of serum levels of the natriuretic peptides and their N-terminal cleavage fragments have diagnostic and prognostic value in patients with known or suspected heart failure. Infusions of nesiritide, a recombinant human BNP, improve hemodynamics and functional status in patients with decompensated heart failure, although the long-term safety and efficacy have been questioned 12, 13 ; . The natriuretic peptides prevent cardiac myocyte hypertrophy in vitro 14 ; . They also affect cardiac mass and left ventricular hypertrophy in animal models. In the spontane * Editorials published in the Journal of the American College of Cardiology reflect the views of the authors and do not necessarily represent the views of JACC or the American College of Cardiology. From the Cardiovascular Institute, University of Pittsburgh, Pittsburgh, Pennsylvania.
BERLIN - Probably all of us who write professionally about musical events have a private little list of personal favorites we feel have for whatever reason not received the acclaim those artists so richly deserve. Why, comparatively speaking, do they remain in the shadow of colleagues no more than their peers - and not infrequently their inferiors? High up on my own little list has long stood the name of the Montral-born, Philadelphia-based pianist Marc-Andr Hamelin. At 45 he does indeed enjoy an international career that definitely demands the adjective "going"; he also has a long and exceptionally congenial relationship with one of the world's musically most distinguished record labels, England's Hyperion. He has made more CDs, many of them esoteric in the extreme, than you could shake several sticks at. Berlin as a matter of course provides the fortunate audience here with the cream of the world's artists, usually at annual intervals. When Hamelin appeared here on Dec. 3 -- at 11 a.m., for the second time only and not even in a conventional concert hall -- I had to ask myself for the umpteenth time: Why not more, much more? Hamelin made his Berlin debut several years ago as one of several younger pianists in a concert series sponsored by the Berlin Philharmonic and presented in the smaller auditorium of the Philharmonie. The visitor I took with me had studied with Nadia Boulanger for seven years, headed the music faculty of a distinguished east-coast college, and definitely, to quote a favorite phrase of Van Cliburn's, knew where middle C was. Details of what Hamelin overwhelmingly laid on us have faded into memory, but I do recall the unfailing musicality that pervaded everything on that entire program. At the end I remarked to my neighbor with something approaching awe: "Well, I'd never expected to hear another pianist who for sheer technical virtuosity outclassed even Horowitz, but I really think I just have" - and my friend agreed. This more recent but still only second Berlin recital by Hamelin came about as a result of his having received one of Germany's highest annual musical accolades, the Schallplattenpreis Recording Prize ; awarded by a jury of some of the country's most knowledgeable and responsible critics. When one of them -- the Frankfurter Allgemeine Zeitung's Dr. Eleonore Bning -- greeted me there, I looked forward to reading her review, for she most definitely also knows where to find middle C. This recital, with conversational interspersings, took place in the main hall of Berlin's Instrumentenmuseum, which houses a splendid collection of instruments of every description from all periods of history and alefacept.
5 DAYS before the procedure STOP all blood thinning products: STOP: Aspirin and aspirin type products, Ginko Biloba, or Vitamin E, Ibuprofen for example: Motrin, Aleve, etc ; May take Tylenol STOP: Anticoagulants --Coumadin Warfarin ; , Heparin or any other blood thinners ; --CHECK WITH YOUR PRIMARY CARE PHYSICIAN TO MAKE SURE IT IS OK OFF YOUR BLOOD THINNERS THAT LONG. STOP: Anti-Platelet medications: Plavix, Pletal, Ticlid or Aggrenox dipyridamole ; products-- CHECK WITH YOUR PRIMARY CARE PHYSICIAN TO MAKE SURE IT IS OK OFF YOUR BLOOD THINNERS THAT LONG At least 2 days prior to your procedure you will need to purchase 3 oz. of Fleet Phospho-soda and 2 dulcolax tabs over the counter at any drugstore. Sold in different amounts Sold in 1.5 oz. bottles 45 ml ; , 3 oz. bottles 90 ml ; , or even 15 ml bottles. Just make sure you get a total of 3 oz. or 90 ml. Recommend purchasing two of the 1.5 oz bottles if your pharmacy has it. PLEASE READ THE FOLLOWING INSTRUCTIONS --YOU MAY NEED OR WANT TO TAKE THE AFTERNOON OFF THE DAY BEFORE YOUR PROCEDURE! The fleet phospho-soda prep will cause diarrhea to clean your colon out for the next days exam ; anywhere from within 15 minutes to 3 hours after taking it. You will need to be close to a bathroom--you may want to take the afternoon off from work the day before to be in the privacy of your own bathroom after taking the prep Fleets Phospho-soda ; or you MAY CHOOSE the alternative prep times see below ; if you do not wish to take the afternoon off the day before your procedure or if you are scheduled for afternoon procedure ; DAY OF EXAM DAY BEFORE EXAM Nothing to eat or drink after midnight Take your normal medications except as noted above. May take heart , blood pressure, and thyroid medications with a CLEAR LIQUIDS ONLY. NO SOLID FOODS. Use liquid diet suggestions below. May drink all the clear liquids you desire all small amount of water at least 2 hours before the procedure day, the more you drink the better your prep will work. For an afternoon procedure, you may drink CLEAR LIQUIDS No red or purple ; up until 4 hours before the procedure 12 NOON FLEET PHOSPHO-SODA PREP TIMES: 12 Noon-- Take first dose of Fleet Phospo-soda SEE Diabetics-- DIRECTIONS BELOW ; Insulin dependent--NO INSULIN or may take of normal 3 Take two 2 ; dulcolax bisacodyl ; tablets swallow ; insulin dose depending on blood sugar--If any questions 6 Take second dose of Fleet Phospho-soda SEE about insulin dose or if not well controlled call your diabetes physician for his recommendations for insulin amounts. DIRECTIONS BELOW ; Oral Medication--Do not take OR ALTERNATIVE FLEET PHOSPHO-SODA TIMES: From check in time to dismissal can be any where from 2 to 12 Noon-- Take two 2 ; dulcolax bisacodyl ; tablets 4 hours depending on difficulty of cases that day. Driver swallow whole ; Expect a bowel movement within does not have to stay but we must have a phone number. Driver approximately 6 hours. must come up to the GI Lab to sign patient out for dismissal If 6 -- Take first dose of Fleet Phospho-soda SEE you want the physician to talk to your family or driver after the DIRECTIONS BELOW ; procedure; please have family or driver stay with you-- because of Morning of procedure --Take 2nd dose of Fleet the sedation medicine you may not remember what the physician Phosphosaid after the procedure ; . Soda 4 hours before scheduled procedure time --That may mean getting up as early as 3 --May drink clear liquids up to 2 hours before the procedure. afternoon procedures 4 hrs ; Clear Liquids: Water, tea, or coffee No milk or non-dairy creamer ; Soft drinks- diet or regular orange, ginger ale, Sprite, 7-Up, etc. No Coke or colored pop ; Gatorade, Kool Aid No red or Purple ; Juices without pulp apple, white grape, lemonade, etc. No Cranberry or Regular Grape ; Soups: Low sodium chicken or beef bouillon broth No noodles or solids with soups ; Desserts: Jello lemon, lime or orange; no fruit or toppings ; No red or Purple Popsicle No sherbets No fruit bars and No Red or Purple ; How to take Fleet Phospho-Soda Mix the Fleet Phospho-soda with glass of a cool clear liquid of your choice. DO NOT HAVE TO DRINK IT FAST-may sip on it over 1 2 hour, esp. if you are becoming nauseated. Drink and follow with at least TWO 8 oz glasses of clear liquid. The more liquids during the day the better. NEED TO TAKE 1.5 OZ 45ML OR 3 MEASURING TABLESPOONS ; for each dose: If you have purchased the 3 oz size bottle--drink for the first dose save the second half for 2nd dose If you have purchased the 1.5 oz size bottle 45ml ; use one bottle for the first dose and the second bottle for the second dose If you have purchased the 15 ml size bottles use three 3 ; for the first dose total 45 ml ; and three 3 ; for the second dose.
Solutions and drugs Rat intracardiac neurons were superfused with physiological salt solution PSS ; containing mM ; : 140 NaCl, 3 KCl, 2.5 CaCl2, 1.2 MgCl2, 7.7 glucose and 10 HEPESNaOH, pH 7.2 or Ca2 + - free PSS containing 1mM EGTA 10 nM free Ca2 + ; . In these neurons, the nicotinic ACh-induced current amplitude was maximal in the presence of 2.5 mM extracellular Ca2 + Fieber and Adams 1991 ; . Bath solutions containing drugs used in a series of experiments were prepared daily. The pipette solution for perforated patch experiments contained in mM ; : K2SO4, 55 KCl, 5 Mg SO4 and 10 HEPES, titrated with N-methyl-D-glucamine to pH 7.2. Amphotericin B-containing solutions were prepared daily and kept on ice and light protected. The osmolarity of all solutions 290 - 310 mmol kg ; was monitored with a vapour pressure osmometer Wescor 5500, Logan, UT ; . Agonists were applied to cells by brief pressure ejection 10 psi; Picospritzer II, General Valve Corp., Fairfield, NJ ; from an extracellular micropipette 3 - 5 m diameter ; positioned 50 - 100 m from the cell soma to evoke maximal responses to agonists. Maximally effective agonist concentrations 300 M ; determined from ACh dose-response relations for [Ca2 + ]i increases, were used for cholinergic receptor activation. To minimise receptor desensitization, a delay of 100 s between agonist applications was maintained. All and aleve.
8 8-MOP A ABILIFY ACCOLATE ACCUZYME acetaminophen codeine acetazolamide ACETIC ACID acetic acid hydrocortisone acetylcysteine ACTONEL ACTONEL WITH CALCIUM ACTOPLUS MET ACTOS ACULAR acyclovir acyclovir sodium ADAGEN ADDERALL XR ADRENALIN ADVAIR DISKUS ADVAIR HFA AGENERASE AGGRENOX albendazole albuterol ALDARA ALDURAZYME ALINIA ALLEGRA-D allopurinol ALOCRIL ALOMIDE ALUPENT AMANTADINE AMBISOME AMERGE aminophylline amiodarone amitriptyline amlodipine besylate amoxapine amoxicillin AMPHOTERICIN B ampicillin ANDRODERM ANDROGEL ANTABUSE ANTHRALIN antibiotic ear 11 9 15 ANUSOL-HC ANZEMET apidra APTIVUS ARANESP ARAVA ARICEPT ARIMIDEX ARIXTRA AROMASIN ARTHROTEC ASACOL asparaginase aspirin ASTELIN ATACAND atenolol ATRIPLA ATROVENT AUGMENTIN AVALIDE AVANDAMET AVANDIA AVAPRO AVODART AVONEX AYGESTIN azathioprine azithromycin B baclofen BACTROBAN BARACLUDE beclomethasone dipropionate benazepril benazepril hcl and hydrochlorothiazide benzocaine benztropine mesylate betamethasone dipropionate betamethasone valerate BETASERON betaxolol hcl brimonidine tartrate brinzolamide bromocriptine mesylate budesonide BUPHENYL bupropion bupropion sr BUSPAR 15 12 9 busulfan butenafine butorphanol BYETTA C CABERGOLINE 13 CADUET 10 calcitriol 13 CAMPRAL 1 CAMPTOSAR 8 CAPITROL 12 captopril 10 captopril hctz 10 CARAC 12 carbachol 14 carbamazepine 6 CARBATROL 6 carbidopa levodopa sr 9 carisoprodol 15 carmustine 8 CASODEX 13 CEENU 8 cefadroxil 6 cefazolin 6 cefixime 6 CEFTIN 6 CELEBREX 6, 8 CELESTONE 12 CELEXA 7 CELLCEPT 14 cephalexin 6 CEREBYX 7 CEREDASE 12 CEREZYME 12 chlorambucil 8 chlorhexidine gluconate 11 chlorpheniramine maleate 15 chlorpheniramine pseudoephe 15 drine chlorpromazine 9 cholestyramine 10 CILOSTAZOL 10 CILOXAN 14 cimetidine 12 CIPRO HC 14 CIPRO I.V. 6 CIPRO XR 6 CIPRODEX 14 ciprofloxacin 6, 14 cladribine 8 CLARINEX 15 8 12.
Reply: Enhancement of embryo developmental potential by a single administration of GnRH agonist at the time of implantation Sir, We have read with interest the letter by Hannam concerning our previously published paper on a beneficial effect of a single administration of a short-acting GnRH agonist to donor oocyte recipients 6 days after ICSI Tesarik et al., 2004 ; . The letter by Hannam describes preliminary experience with the same protocol applied in ovarian stimulation cycles and suggests that the treatment with GnRH agonist in the luteal phase may increase the risk of ovarian hyperstimulation syndrome OHSS ; . In this comment we should like to summarize the recent clinical data regarding this new indication for GnRH agonist. In the aformentioned prospective randomized study Tesarik et al., 2004 ; we used a design based on an oocyte-sharing oocyte-donation programme. This design was chosen to evaluate the potential direct effect of GnRH agonist on the implanting embryo while excluding parallel effects on the corpus luteum. In fact, this structure was absent in the oocyte recipients receiving the luteal-phase GnRH agonist treatment, either because of their spontaneous anovulatory status or as a sequela of previous pituitary down-regulation. Hence, ovulation was absent, and the luteal phase was maintained artificially by exogenous progesterone administration. Consequently, the risk of OHSS was non-existent. However, extension of these findings to other clinical scenarios, including those involving controlled ovarian stimulation protocols, was a logical next step to be explored. Hannam reports cases of mild ovarian hyperstimulation syndrome OHSS ; after luteal GnRH agonist administration as well as one case of severe OHSS which was, however, completely resolved within 48 h. This latter observation was made in an at-risk patient. The effects of GnRH agonist administered in the luteal phase remain a controversial issue. Early studies suggested that GnRH agonist treatment in the luteal phase 1 or 2 administrations of 500 g buserelin ; may act as a luteolytic agent for contraceptive purposes Lemay et al., 1982, 1983 ; , supposedly due and alfuzosin.
The Wild Flower Page Habitat wildflwr Natural History Museum nhm.ac The Wildlife Trusts national website with links to local Wildlife Trusts sites wildlifetrust Woodland Trust woodland-trust British Bryological Society rbge bbs British Mycological Society ulst.ac faculty science bms Ancient Tree Forum woodland-trust ancient-tree-forum Cornish Wildlife Group groups.yahoo group cornishwildlife Flora locale floralocale National Biodiversity Network nbn National Federation for Biological Recording nfbr Natural History Book Service nhbs Sedge Identification Key keyhomepages.ed.ac ebfr89 key keys Bryophyte pictures pictureshome.clara adhale bryos phframe Flora keys reticule flora Phenology website woodlandtrust phenology newsletter Rare plants discussion group groups.yahoo group ukrareplants UK Flora writers smartgroups groups Florawriters Launceston Parish Wildlife Project parish-wildlife index.
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1 2 normal saline .T-56 8-MOP.T-39 aa 4.25% calcium lytes d25w .T-35 aa 4.25% electrolyte-tpn d10w .T-35 ABELCET.T-16 ABILIFY.T-53 ABILIFY DISCMELT.T-53 ABRAXANE .T-25 ACCOLATE .T-21 Accupril.T-55 Accuretic .T-55 Accutane .T-59 acebutolol hcl.T-33 acetaminophen with codeine.T-3 Acetasol-Hc.T-19 acetazolamide .T-17 ACETAZOLAMIDE SODIUM.T-17 acetic acid .T-18 acetic acid aluminum acetate .T-18 acetic acid hydrocortisone.T-19 acetylcysteine .T-49 Achromycin V.T-11 Aclovate .T-22 ACTHIB.T-62 Actigall.T-39 ACTIMMUNE.T-47 Actiq.T-3 ACTIVELLA .T-42 ACTONEL.T-47 ACTONEL WITH CALCIUM .T-47 ACTOPLUS MET .T-15 ACTOS .T-15 ACULAR .T-21 ACULAR LS .T-21 ACULAR PF.T-21 acyclovir.T-32 acyclovir sodium .T-32 ADACEL .T-61 ADAGEN.T-42 Adalat Cc .T-35 Adapin.T-52 Adderall.T-5 ADDERALL XR .T-5 Adoxa.T-11 ADOXA PAK .T-11 Adriamycin .T-26 Adrucil .T-26, T-59 ADVAIR DISKUS.T-60 ADVAIR HFA .T-60 ADVICOR .T-24 AEROBID.T-1 AEROBID-M.T-1 AGENERASE.T-30 AGGRENOX .T-63 Agrylin .T-47 AKINETON.T-11 ALAMAST .T-6 Albalon.T-63 ALBENZA.T-6 albuterol.T-60 albuterol sulfate .T-60 alclometasone dipropionate.T-22 Alcohol In Dextrose.T-36 ALCOHOL SWABS.T-20 Aldactazide .T-55 Aldactone .T-55 ALDARA.T-58 Aldoril .T-45 ALDURAZYME.T-42 Alesse.T-39 ALFERON N .T-32 ALIMTA .T-25 ALINIA.T-29 ALKERAN .T-25 Allegra.T-57 ALLEGRA-D 12 HOUR .T-57 ALLEGRA-D 24 HOUR .T-57 allopurinol.T-47 allopurinol sodium .T-47 ALOCRIL .T-21 ALOMIDE .T-6 Aloprim .T-47 ALORA.T-42 ALOXI .T-15 Alphagan .T-41 ALPHAGAN P .T-41 Alphatrex.T-22 ALTABAX .T-19.
Phenylketonuria PKU ; : 1 in 10, 000 live births about 10 babies per year ; . PKU causes high blood levels of phenylalanine and severe intellectual disability. A diet low in phenylalanine, started in the first two to three weeks results in normal development. Primary congenital hypothyroidism: 1 in 3, 500 live births about 26 babies per year ; . It is caused by the absence or abnormal formation or function of the thyroid gland. This causes growth and intellectual disability if not treated. Medication with thyroid hormone started early, results in normal growth and development. 58 12 06 and allergen.
Vintage Crime Black Lizard, a division of Random House paperback ; 2002. 308 pages. .00 Review by Donald R. Wesson, MD FROM JUST THE TITLE, one might think that this is a textbook for physicians, but it's not. It's a novel in the genre of crime fiction. A subplot involves a heist of a fictional new opiate, Ultracept, which a group of friends and relatives of pain patients are planning to make available outside medical channels for treatment of patients with intractable pain. The members of the renegade group have one thing in common: they've all watched someone they cared deeply about suffer excruciating pain. A woman describes the treatment of her younger brother before she took matters into her own hands.
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The Guide recommends 96.8 cm2 per mouse as an appropriate housing density. Yet scientific validation of these recommendations is lacking. The purpose of this study was to evaluate the effects of cage size and environmental enrichment on some physiological parameters potentially indicative of stress responses. Female mice from 3 strains C57BL 6NCrl, BALB cAnNCrl, and Crl: CD1 ICR were housed in polycarbonate cages each containing 4 same-strain mice under 1 of the following conditions: small 58 cm2 per mouse ; unenriched cage, small enriched cage, medium 96.8 cm2 per mouse ; unenriched cage, medium enriched cage, large 219.4 cm2 per mouse ; unenriched cage, and large enriched cage. There were 5 replicate cages per treatment, and commercially available enrichments for example, nestlets, tunnels ; were used. After 12 to 16 wk, the mice were euthanized by carbon dioxide asphyxiation, blood collected by cardiocentesis for white blood cell WBC ; counts, and a complete necropsy performed. Thymus, spleen and adrenal weights were recorded. Statistical analysis was performed using MANOVA. The was no effect of enrichment on WBC counts, but there were significant interactions between strain and cage size for total WBC counts F4, 86 5.02, P 0.001 ; , lymphocytes F4, 86 3.352, P 0.014 ; , and monocytes F4, 86 3.05, P 0.02 ; . In C57BL 6 and CD-1 mice total WBC counts and lymphocyte levels showed a U-shaped curve, being lowest in the medium-sized cages. The curve for monocytes in CD-1 mice was similarly U-shaped, but in C57BL 6 monocyte counts were highest in the smallest cages. In contrast, in BALB c mice, total WBC counts, lymphocyte, and monocyte counts all decreased linearly with decreasing cage size. While there were significant differences in some organ weights between strains, there were no effects of enrichment or density on organ weights. These results show that cage size affects leukocyte profiles that may be indicative of a stress response, and that these effects are strain dependent and almotriptan.
Pham, T; Azulay-Parrado, J; Champsaur, P; Chagnaud, C; Legre, V; Lafforgue, P. "Occult" osteoporotic vertebral fractures - Vertebral body fractures without radiologic collapse. SPINE 30 21 ; . NOV 1 2005. p.2430-2435 Patients who presented with osteoporotic vertebral fractures with no visible deformation of vertebral body, presenting with acute back pain with no initial deformation of the vertebral body on plain radiographs, and later proved to be fresh osteoporotic vertebral body fractures were studied. All cases showed the incriminated vertebra appeared normal on initial radiographs, the diagnosis of fresh vertebral body fracture was confirmed by MRI, and the diagnosis of osteoporosis was made by the combination of established osteoporosis, ruling out of underlying disease, and follow-up. 21 fractures in 16 patients 11 female 5 male; mean age, 72 years ; were found. Most of these fractures affected the lumbar spine 14 of 21 occurred at L2 - L5 ; Osteoporosis was known beforehand in 9 patients and newly diagnosed in 7 patients. At follow-up, radiographs were obtained for 19 of 21 fractures: in 15 cases, the vertebral fracture developed a vertebral collapse in a mean of 12.5 weeks range, 4 - 24 and aggrenox.
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